Morning stiffness
Stiffness lasting more than 30–60 minutes, especially after waking or a long period of rest.
Persistent joint swelling is not “just age”. A careful examination, the right blood tests and early disease-modifying treatment can protect joints and everyday function.

Separate pages with simple dropdown answers. Use the results for discussion with your clinician, not for self-diagnosis.
A simplified 2010 ACR/EULAR score with both clinical safety checks. The detailed calculator remains below.
Open RA assessment →Back or buttock painExplore the inflammatory back-pain pattern, supporting symptoms and investigations a clinician may consider.
Open AS self-check →Dry eyes and dry mouthReview the five 2016 ACR/EULAR criteria, clinical checks and your available test results. Share each item’s points with the clinic.
Open Sjögren’s assessment →Skin, joints and systemic symptomsReview the 2019 EULAR/ACR criteria, with ANA entry, clinical safeguards and one score per domain.
Open lupus assessment →Psoriasis, nails and joint symptomsExplore the CASPAR criteria using confirmed inflammation, skin and nail findings, and test results.
Open psoriatic arthritis assessment →Sudden painful joint attacksReview the 2015 ACR/EULAR criteria, including crystals, blood urate and imaging, with a detailed point breakdown.
Open gout assessment →Tight skin and finger colour changesReview systemic sclerosis criteria, clinical eligibility, skin changes and supporting investigations.
Open scleroderma assessment →Widespread pain, fatigue and sleepRecord pain areas, symptom severity and duration using the 2016 revised symptom criteria.
Open fibromyalgia assessment →One symptom alone does not prove RA. The pattern, duration, true joint swelling and examination findings matter together.
Stiffness lasting more than 30–60 minutes, especially after waking or a long period of rest.
Persistent puffiness, tenderness or warmth—often in the knuckles, finger joints, wrists or feet.
Similar joints may be affected on the right and left, though early disease is not always perfectly symmetrical.
Unusual fatigue, low energy, reduced appetite or a low-grade fever can accompany joint inflammation.
if swelling or morning stiffness persists for several weeks, small joints are involved, or daily tasks such as making a fist and buttoning clothes are becoming difficult.
Enquire nowThere is usually no single cause. RA develops through a combination of immune, genetic and environmental factors.
RA is an autoimmune disease. The immune system attacks the lining of the joints, creating ongoing inflammation.
Family history and certain genes can increase risk, but inheritance alone does not determine who develops RA.
Long-term smoking is a well-established risk factor. Age, sex and hormonal factors also influence risk.
There is no single test that “proves arthritis”. Tests answer different questions, and their meaning depends on the pattern of symptoms and examination.
When symptoms began, whether onset was sudden or gradual, and whether morning stiffness lasts beyond 30–60 minutes.
Which joints are swollen—not only painful—and whether small joints, both sides, the neck or lower back are involved.
Difficulty making a fist, opening jars, dressing, walking, working, sleeping or managing daily activities.
Fatigue, fever, weight change, dry eyes/mouth, rashes, psoriasis, mouth ulcers, bowel symptoms or eye inflammation.
Family history, smoking, recent infection, pregnancy plans, previous medicines, allergies and other medical conditions.
Rheumatoid factor is an antibody found in many people with RA. It can also occur with other illnesses, infections, increasing age—or in someone who never develops RA.
Helpful, but not a yes/no test. RF can be positive without RA, and some people with RA remain RF-negative.
More specific for RA than RF and can become positive early, but a negative result still does not completely exclude RA.
Show the level of inflammation and help monitor response. They can be normal in some people with active RA.
Check anaemia, baseline health and whether planned medicines can be used and monitored safely.
X-rays may be normal early. Ultrasound or MRI can show synovitis before permanent damage is visible on X-ray.
No blood test proves RA alone. A clinician must confirm true synovitis and consider other causes of joint symptoms.
Add the four domains. A total of 6/10 or more reaches the classification threshold—but only after the two clinical entry requirements are met.
These are classification criteria, not a home diagnostic test. They apply only when a trained clinician has confirmed synovitis in at least one joint and another disease does not better explain it.
A score alone cannot be interpreted until both clinical safety gates are confirmed.
Prefer dropdown answers? Open the separate RA assessment page →
RA treatment is individualised and adjusted to a target—usually remission or low disease activity.
DMARDs act on the disease—not just pain. Methotrexate is a common anchor medicine; hydroxychloroquine, sulfasalazine or leflunomide may suit selected patients.
If standard DMARDs do not reach the treatment target, biologic or targeted synthetic DMARDs may be considered after appropriate screening.
NSAIDs and carefully limited corticosteroids may reduce pain and inflammation while disease-modifying treatment begins to work. They do not prevent damage by themselves.
Graded exercise, physiotherapy, occupational advice, sleep, smoking cessation, vaccination and cardiovascular/bone-health review support long-term outcomes.
A clinician may aspirate fluid and/or inject corticosteroid into a selected swollen joint to reduce inflammation quickly. Ultrasound guidance may be used for accuracy in some joints.
Modern treatment can bring many people into remission or low disease activity, reduce symptoms and prevent damage. Even when you feel well, ongoing monitoring helps keep the disease controlled.
A balanced eating pattern rich in vegetables, fruit, whole grains, pulses, nuts and suitable protein supports overall health. It is not a substitute for RA treatment.
No single everyday food has been shown to cause RA. Avoiding multiple foods without a clear reason can lead to poor nutrition.
Diet can support overall health, but it does not replace DMARD treatment. Be cautious with unlabelled herbs and supplements.
Gluten restriction is not routine for RA. It is relevant when coeliac disease or another clear intolerance has been diagnosed.
Pain varies for many reasons. Keep a simple record and discuss reproducible patterns before making major restrictions.
False. RF must be interpreted with symptoms, examination, anti-CCP, inflammation tests and alternatives.
False. Seronegative RA exists, and diagnosis remains clinical.
Appropriate, graded exercise helps maintain strength and function. The plan should be adjusted during an active flare.
These may ease symptoms, but only a complete disease-control plan addresses the underlying immune inflammation.
Do not stop DMARDs on your own. Symptom relief can occur before inflammation is fully controlled.
RA is autoimmune and can occur at many ages. Osteoarthritis and RA have different patterns and treatment.
Dr Ranjeet Kaur · RheumatologyMBBS · MD (Medicine) · Fellowship in Rheumatology
Specialist assessment and medical management for inflammatory arthritis, autoimmune rheumatic conditions and long-term joint health—with a focus on careful diagnosis, disease control and preserving function.



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